The advantage of Western medicine in the treatment of uremia from kidney function, scientific research and analysis objectively and carefully, and pay attention to the actual root cause tracing. In addition, great attention has also been paid to the human pathological reactions caused by microorganisms such as bacterial toxins.
1, through the control of blood pressure to achieve renal protection. In recent years, angiotensin converting enzyme inhibitors (ACEI) and angiotensin Ⅱ receptor antagonist (Ang Ⅱ RA) development and application of renal hypertension treatment and by lowering Pressure to achieve kidney protection to create a new milestone, ACEI and Ang Ⅱ RA has been recognized as a delay in chronic renal failure progression of the role.
2, erythropoietin (EPO) treatment of renal anemia. In the mid-20th century, the mid-80s recombinant human EPO and the creation of a new milestone in the field of renal disease for the treatment of renal anemia. Winearls et al first reported in 1986 that the use of recombinant human EPO for the treatment of 10 renal anemia patients was successful, and later large samples were also satisfactory results, which became a breakthrough in the treatment of renal anemia. EPO is the use of DNA recombinant count of synthetic hormone, its biological activity, immunological characteristics and natural EPO exactly the same, no obvious side effects.
3, low-protein diet (LPD) or LPD + essential amino acid therapy (EAA) or LPD + α-keto acid therapy LPD delayed CRF progression has been recognized in the 20th century, 80 years, 90 years very stressed. It is generally believed that protein intake in the 0.5 ~ 0.6g / kg • d can maintain the patient's nitrogen balance. LPD + EAA or alpha-keto therapeutics, making it possible to strictly limit the protein and avoid hypo-hypersensitivity to malnutrition. In recent years, the choice of dietary protein source has a new understanding, so that the monotonous diet of CRF patients persified, such as fear of BUN increased ban on legumes, that is unfounded.